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By Karen Collins, MS, RD, CDN - Q: Will I get more nutrition from eating carrots raw or cooked? Q: I've cut back on meals to try to lose weight, but I still snack. I don't seem to be losing weight so do I have to cut my snacks too?

A: It might be your snack choices that are holding you back. The latest national survey of U.S. eating habits shows that on average, about a third of the "empty calories" we eat come from our snacks. Empty calories are calories we get from foods that supply little if any nutrients or protective plant compounds. Many of the foods Americans typically choose for snacks come with a high calorie load in a relatively small portion. If snacks are part of your weight control challenge, consider nutritional, behavioral and psychological solutions. If you need only a small snack to tide you over to your next meal, fruit or raw vegetables would be a great choice instead of chips or sweets with calories that add up so quickly. For a snack that will sustain you longer, add a little protein, such as some yogurt or a handful of nuts. Perhaps the problem involves how you snack: if you sit down with the whole bag of chips or cookies, chances are that despite intentions to eat just a bit, you will eat more than you intended. Whatever you choose, take out the amount you think you need and put the container away. Another possibility is that you are using snacks to treat yourself or cope with stress and getting loads of empty calories your body doesn't need. Look for other ways to unwind that are truly being good to yourself, like taking a break for a brief walk around the block on a beautiful day or even a few moments of deep breathing to decompress. If none of these snacking problems is the issue, check your overall eating habits using the free online MyPlate Food Tracker. If you're not clear about how to make workable changes, find a registered dietitian who can help you individually to develop a strategy.

Q: Will I get more nutrition from eating carrots raw or cooked?

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HealthTalk: Cooked vs Raw Carrots and Snack Choices

Running back Trent Richardson (3) was a coveted five-star recruit in 2009, while corner Morris Claiborne (17) was a three-star prospect who thought he'd play receiver.

Matthew Emmons/US PRESSWIRE

NFL Draft Preview

First-round QB busts

Past draft bargains

Historic draft moments

No. 1s through the years

If Peter King picked the first round correctly in his Mock Draft last week, nearly half of this year's newest crop of millionaires will have played high above recruitniks' predictions. Fifteen of the 32 players in King's Mock rated three stars or fewer out of high school, meaning evaluators felt they would, at best, be solid contributors at the FBS level.

So how did the recruitniks misjudge so many players so badly? They didn't. They misjudged a few players, which is to be expected when trying to project how 17-year-olds will fare as 20-year-olds. After last year's draft, writer Matt Hinton broke down the numbers, and they backed up the star ranking system's relative reliability as a predictor of success in college and beyond.

If 15 of 32 earning three stars or fewer sounds like a lot, consider the fact that from 2003-08, Rivals.com ranked 208 players as five-stars, 1,807 players as four-stars and 13,862 as a three-star or lower. In other words, two- and three-star players made up 87.3 percent of the players Rivals ranked during that period. Meanwhile, four- and five-stars -- of which King's Mock included 17 -- made up only 12.7 percent of the players ranked during that period. So, if the numbers hold, 53 percent of the first-rounders will come from the top eighth of the recruits.

Read more from the original source:
Andy Staples: How top 2012 NFL Draft prospects ranked as high school recruits

April 18, 2012 Updated Apr 18, 2012 at 8:25 AM EDT

UNDATED (Indianas NewsCenter) Nose feeding tubes are typically used on patients who are suffering from head or neck cancer and cannot swallow but now they are helping some to shed pounds quickly.

Called the K-E Diet, which is like an extreme form of the Atkins Diet.

Where most people lose about 2 pounds per day for 10 days.

Heres how it works: you're fed continuously through a feeding tube, small spaghetti sized tube, which gets inserted through the nose under local anesthesia."

It's slightly uncomfortable. And the patient has to carry a feeding pump 24 7.

Mixed with water, that's all the nourishment the patient receives, an infusion of proteins and fats, and no carbohydrates. The patient's intake is only 800 calories a day. But the infusion is constant and there are zero carbohydrates, which curbs hunger.

The body rapidly goes into profound ketosis and when you're in ketosis the body starts to burn fat not muscle which is why you lose so much weight so quickly. The diet started in Rome about 10 years ago, physicians began using feeding tubes on their patients as a way to lose weight. Then last July, the diet was brought to the United States to help the morbidly obese. But then he saw a shift in demand, including from brides to be.

Slowly the need and the demand shifted toward mainly towards women that needed to lose weight for a specific situation. Brides began coming in and saying, "I need to fit into my wedding dress because I'm a nervous eater, and I've been gaining weight."

But not everyone thinks the diet is a good idea.

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Extreme Dieting: Brides-to-Be Using Feeding Tubes to Rapidly Shed Pounds

SAN DIEGO (TheStreet) -- Last week's column on Vivus ARNA and Arena Pharmaceuticals VVUS drew some strong reactions from the Arena faithful, almost all of whom were spitting mad that I dared say publicly what almost everyone else already believes: Arena's weight-loss drug lorcaserin isn't going to be approved any time soon. FDA, however, is getting ready to approve Vivus' Qnexa. In particular, the "Areniacs" accused me of lying about lorcaserin and being paid off to write disparaging, false and misleading statements about Arena by short sellers and other criminal market manipulators. This paragraph, in particular, raised a fuss: "Lorcaserin isn't much more than a placebo (a paltry 3% weight loss) with the added bonus of causing tumors in rats. Arena's has failed to disprove a link between the lorcaserin-fueled rat tumors and the potential for breast cancer in humans, despite the company's claims to the contrary. And let's not forget the risk of heart valve damage that still lingers over lorcaserin, a chemical cousin to the infamous and dangerous fenfluramine." A personal injury lawyer from Michigan was so upset that he got together with a bunch of Arena retail shareholders -- financially conflicted all -- and wrote me a letter demanding a retraction to my "terrorist style hit-piece sic designed to put lorcaserin 10 mg BID in false light." 10 Stocks That Could Rise in Market Decline >> Apparently, writing a column predicting the rejection of a weight-loss drug because it doesn't help people lose weight and is potentially unsafe is a terrorist act. A retraction is not going to happen, but I will follow up and explain more fully why I believe FDA isn't going to approve lorcaserin based on the data available. Some cautious words from Arena's recently filed 10-K to get started: "We expect the FDA to conduct a benefit-risk assessment based upon the totality of the new and previously provided data to determine the approvability of lorcaserin. It is important to note that the FDA may analyze or weigh the data differently than we or others do. (Emphasis mine.) "Others" include Arena bulls who post obsessively on stock message boards. More warnings from Arena: "In addition, the analyses we included in the resubmitted lorcaserin NDA include estimates based on certain assumptions and extrapolations. The FDA may accept our assumptions and extrapolations or may use different ones in analyzing the data, which could lead the FDA to reach different conclusions. (Emphasis mine.) 4 Stocks That Are Real Sleepers in 2012 >> We already know FDA reached a "different conclusion" about lorcaserin during the first review. The drug was rejected. In doing so, FDA raised a number of safety issues and concerns focused mostly on lorcaserin causing tumors in rats and the potential risk for similar tumor formation (including breast cancer) in humans. Arena re-analyzed old rat data and conducted some new rat studies to collect more data to alleviate the FDA's concerns about lorcaserin-fueled tumor growth. Arena bulls are convinced the new data exonerate lorcaserin. I look at the same data and walk away believing the opposite. At best, these new data are inconclusive i.e. the data neither confirm nor reject lorcaserin's link to tumor formation. That's not good, and coupled with concerns about lorcaserin's potential to cause heart valve damage and weight-loss efficacy not much greater than placebo, FDA will reject lorcaserin a second time. Let's take a look at the evidence to support the "lorcaserin is not approvable" thesis: From Arena's 10-K: "In rats given lorcaserin, the incidences of certain tumors, including benign and malignant tumors of the mammary gland, were increased relative to placebo. In female rats, the specific diagnoses (i.e., benign or malignant) differed between an initial pathologists analysis of the tissues on an ongoing basis during the study and the final peer-reviewed diagnoses reported in the study report and included in the original NDA. The FDA recommended that the tissue samples be re-adjudicated to provide greater certainty for the diagnoses of benign and malignant mammary tumors. "We convened a pathology working group, or PWG, that consisted of five independent veterinary pathologists to re-adjudicate the female rat mammary tumor diagnoses in the rat carcinogenicity study. The FDA reviewed and agreed to the PWG membership and procedures." Let's stop here for a second to address the mistaken notion that that FDA chose the pathologists to sit on the PWG, or otherwise endorsed the group's finding. That's false. Arena chose, hired (and most importantly) paid for the pathologists. FDA may not have objected to the makeup of this group, but make no mistake, these pathologists work for Arena. With that said, I'm happy to endorse the independence and unbiased diligence of the PWG. After re-examining the slides of rat tumor samples from Arena's original animal studies: "The PWG concluded that the incidence of mammary adenocarcinoma in the high-dose, or 100 mg/kg/day, female group and the increased incidence of mammary fibroadenoma in each of the low-, mid- and high-dose, or 10, 30, and 100 mg/kg/day, respectively, female groups were lorcaserin-related," according to Arena's 10-K. Translated: Lorcaserin at the highest dose caused rat breast cancer (mammary adenocarcinoma). Lorcaserin at every dose studied (low, mid and high), caused benign breast tumors (mammary fibroadenoma) to form. These were not the only tumors that the PWG found in rats after re-examining the old slides. Lorcaserin was also associated with higher rates of mammary adenoma (a benign tumor at all doses; as well as higher rate of carcinosarcoma (cancer) at the high dose. Next, Arena tried to address FDA concerns about the relationship between the lorcaserin dose (low, mid and high) and the "aggressiveness" of rat breast cancer (mammary adenocarcinoma) formation. From Arena's 10-K: "The PWG considered measures of aggressiveness for mammary adenocarcinoma, which included time-to-tumor diagnosis, or latency; number of animals with more than one primary tumor, or multiplicity; and number of animals with mammary adenocarcinoma metastases in, or spread to, the lung. Latency was significantly shortened from control when analyzed as a trend across lorcaserin doses; we performed a post hoc analysis of latency for individual doses that showed only the high dose differed significantly from control." (Emphasis mine.) Translation: Rat breast cancer grew faster in rats treated with lorcaserin compared to rats treated with a control. Arena thinks high-dose lorcaserin is the culprit but that conclusion came only after a "post hoc analysis" was performed. Is this another spot where FDA may do its own analysis and reach a different, more troublesome conclusion? More on the PWG analysis, again from Arena's 10-K: "Multiplicity was greater than control in the high-dose group, but not in the low- or mid-dose groups (10.8%, 9.2%, 9.2% and 22.7% in the control, low-, mid- and high-dose groups, respectively). Translation: Only rats treated with high doses of lorcaserin had more than one primary tumor compared to control. More: "Among all of the animals in each dose group, the incidence of lung metastases from mammary adenocarcinoma was 0%, 1.5%, 7.7% and 6.7% (control, low-, mid-, and high-dose groups, respectively), and, among only the animals with mammary adenocarcinoma in each dose group, the incidence of lung metastases from this tumor was 0%, 4.8%, 20.8% and 9.8% (control, low-, mid-, and high-dose groups, respectively); the PWG described the incidence of lung metastases as low for all groups, with basically no difference between the control and the low-dose group and an equivocal increase in the mid- and high-dose groups. " Translation: Mid- and high-dose lorcaserin is linked to lung metastases, or tumors that spread to the lung. Nonetheless, Arena says, "the PWG reached the conclusion that mammary adenocarcinoma was lorcaserin treatment related only at the high dose." Says the PWG, but what will FDA say given the existence of data linking all doses of lorcaserin to at least some level of tumor aggressiveness? Arena conducted an additional analysis of these rat data, looking at time to death due to tumor formation in these same rats. The findings, disclosed in Arena's 10-K, raise more questions: "Time to death specifically attributed to adenocarcinoma was significantly accelerated only in the high-dose group. In the second analysis, time to death due to unspecified mammary tumor was accelerated in the mid-dose group as well, although, as discussed above, the incidence of adenocarcinoma was not increased over control in the mid-dose group." Question: The incidence of rat breast cancer (adenocarcinoma) may not have been elevated in rats treated with the middle dose of lorcaserin but were these fewer tumors more aggressive and caused the rats to die faster? Arena wants to assure everyone that only high-dose lorcaserin is linked to rat tumor growth, but again, the middle dose of the drug keeps getting implicated. Arena summarizes the work of the PWG in its 10-K: "We believe the PWGs re-adjudication and their and our related analyses found that mammary adenocarcinoma was only lorcaserin treatment related at the high dose, establishing a safety margin of 24-fold between human lorcaserin exposures at the intended therapeutic dose and exposures in rats at the highest dose not associated with malignant mammary adenocarcinoma." (Emphasis mine.) "We believe" doesn't mean, "FDA believes." Arena's "belief" also assumes a clear understanding of how lorcaserin blood levels in rats translate into similar blood levels in humans. Arena provides no additional evidence to demonstrate confidence in that assumption. Up to this point, we've been talking mainly about lorcaserin causing adenocarcinomas, or malignant (cancerous) tumors in rats, but any tumor growth that potentially occurs in humans, even the benign variety, is a concern. Doctors can't necessarily distinguish between benign and malignant tumors without an exam and biopsy, which means additional costs, anxiety and treatment for patients. From Arena's 10-K: "With respect to fibroadenoma benign tumors , the PWG re-adjudication did not establish a safety margin for these benign tumors since they were increased over control at all lorcaserin doses tested. The PWG determined that, in addition to incidence, tumor multiplicity was increased in all lorcaserin groups, and that tumor latency was significantly decreased as a trend across all lorcaserin dose groups. A separate analysis we performed also showed that tumor latency was significantly decreased in each treatment group." (Emphasis mine.) Uh oh... You get this? Arena tells us that lorcaserin, regardless of the dose, caused more benign tumors to form in rats compared to control. These tumors grew fast, emerged sooner and were more frequent. And this is not a problem because...? Moving on to the question of why lorcaserin is causing the growth of tumors in rats. Arena blames prolactin, a reproductive hormone. Lorcaserin increases the effects of prolactin, which in turn, causes evidence of abnormal growth of cells in the rat mammary glands, or so Arena claims. To test the hypotheses, the company conducted new rat studies that compared the prolactin-boosting effects of lorcaserin against perphenazine, a schizophrenia drug known to increase prolactin levels and cause changes in rat breast tissue. Once again, back to Arena's 10-K: "After optimizing methodology in the pilot experiments, we conducted a three-month study of intact female rats. The prolactin content in the pituitary gland was measured after dosing lorcaserin, perphenazine, or control for 7, 28, 60 and 90 days, and plasma prolactin concentrations were measured at various time points throughout the study. Lorcaserin at all doses tested (10, 30 and 100 mg/kg/day, or the low-, mid- and high-dose, respectively) increased pituitary prolactin content relative to control at all time points after 7 days; these increases were significant for all lorcaserin doses at Day 90. Lorcaserin also significantly increased plasma prolactin compared to control at 20 hours after dosing for up to 10 days at the high dose." If Arena's theory about lorcaserin boosting prolactin levels is correct, the drug should act very much like perphenazine, but the data excerpted above are inconsistent. Lorcaserin-treated mice at all doses exhibit increases in pituitary prolactin content relative to control but only the high dose of lorcaserin caused an increase in levels of plasma prolactin compared to control -- and even then, only for a short period of time (10 days), not 28, 60 or 90 days. Did plasma levels of prolactin in high dose lorcaserin-treated mice fall after 10 days? Or did the mice die so quickly that plasma levels couldn't be measured at 28, 60 and 90 days? Arena doesn't say but without the data, this experiment is incomplete and therefore inconclusive. Arena then examined the rat breast tissue to see if any cellular changes could be observed which would hint at increased prolactin levels causing tumor formation. Some, but not all, of these examinations yielded positive results. In particular, Arena notes, "Low-dose lorcaserin significantly increased PCNA staining at Day 90, and mid-dose lorcaserin increased the signal at Day 28. High-dose lorcaserin did not significantly affect PCNA staining in this study. An increase in PCNA staining indicates cellular proliferation." Odd. Arena insists that high dose lorcaserin is the culprit, yet it had not affect on PCNA staining but low- and mid-dose lorcaserin did -- significantly. This is another example of data inconsistency that raises questions because lorcaserin isn't acting quite like perphenazine, even though it should if Arena's theory is valid. Here's how perphenazine performed in this study, as described by Arena: "The positive control, perphenazine, caused significant elevations of plasma prolactin at all time points, more pronounced morphological changes at Days 28 and 90 and significantly decreased pituitary prolactin content at Days 28 and 90. Perphenazine significantly increased lobular structures and decreased terminal ducts in whole mount preparations, significantly increased lobular hyperplasia in H&E sections and significantly increased PCNA immunostaining of mammary tissue on Days 28 and 90." Then there's this Arena 10-K disclosure: "A smaller study in male rats did not show consistent effects on pituitary or plasma prolactin or mammary morphological changes with lorcaserin or perphenazine treatment. Since the positive control had no effect relative to control, this experiment does not contribute to our interpretation of the prolactin hypothesis." Translation: This study in male rats was a total failure so we're just going to make believe it never happened even though it should have shown consistent and similar results to the studies we did in female rats in order for our prolactin-lorcaserin theory to make sense. Enough about rats and tumors, let's move on to the issue of heart valve damage, or valvulopathy. During the first review, FDA and some of the experts at the agency's advisory panel meeting expressed concern about lorcaserin's potential to cause heart valve damage, based on data from Arena's two phase III studies known as Bloom and Blossom. Arena, of course, disagreed, expressing the view that lorcaserin was designed in a way to avoid the valvulopathy issues that caused fenfluramine, a similar drug, to be yanked off the market. Last November, Arena released data from a third, late-stage study known as Bloom-DM, which showed lorcaserin-treated patients with more heart valve problems than patients treated with a placebo. FDA did not have the Bloom-DM data during lorcaserin's first review but Arena included these data in its resubmission. Again, let's go to Arena's 10-K. Pardon the lengthy excerpt: "Data from our two large Phase 3 lorcaserin trials of one and two years in duration, BLOOM and BLOSSOM, including the proportions of patients that developed FDA-defined valvulopathy, were included in our original lorcaserin NDA. There are different ways of analyzing the valvulopathy data from our trials. The pre-specified statistical analysis plan for the NDA provided that the risk difference between lorcaserin and placebo using Baseline and Week 52 echocardiograms would be evaluated using a non-inferiority model that would rule out a greater than 50% increase over the assumed placebo risk of FDA-defined valvulopathy. The assumed placebo risk of FDA-defined valvulopathy was derived from the Data Safety Monitoring Board, or DSMB, interim review of six-month data from BLOOM. Using this analysis, the combined data from BLOOM and BLOSSOM demonstrated that lorcaserin was non-inferior to placebo. Using a relative risk analysis of the Baseline and Week 52 data, which the FDA has used previously and may favor over the above analysis, these trials ruled out an increase of more than 55% in the relative risk for FDA-defined valvulopathy with lorcaserin." (Emphasis mine.) The last sentence, highlighted, is the most relevant. FDA set the safety bar at ruling out a greater than 50% increase in risk for valvulopathy. Arena said its analysis of the Bloom and Blossom studies ruled out a 55% risk, meaning lorcaserin was safe. As I said above, there was discussion and disasgreement from advisory panel members who believed Arena actually fell just short of the valvulopathy safety margin. Okay, but what happens when valvulopathy data from the new Bloom-DM study are added to the mix? Again, here's what Arena discloses in its 10-K: "Our other one-year Phase 3 clinical trial, BLOOM-DM, was not designed to include enough patients to be adequately powered to detect meaningful differences in the incidence of valvulopathy. Rather, we pre-specified a combined analysis of echocardiographic changes in all three Phase 3 trials. We integrated into our lorcaserin NDA resubmission the results of BLOOM-DM, which include additional data relating to heart valves and pulmonary artery pressures. Using the analysis of risk difference that we used for our original NDA, the pooled data from BLOOM, BLOSSOM and BLOOM-DM would rule out a greater than 50% increase over the assumed placebo risk of FDA-defined valvulopathy. Using a relative risk analysis, the pooled data from all three trials ruled out an increase of more than 67% in the relative risk of FDA-defined valvulopathy. Statistical methods that consider all echocardiograms rather than restricting the analysis to Baseline and Week 52 produced risk ratio or hazard ratio estimates of 1.08 -- 1.09, ruling out a 44% increase in risk of FDA-defined valvulopathy. (Emphasis mine.) Discrepancies! Depending on how the pooled data from the three trials are analyzed, lorcaserin either clears the FDA's valvulopathy risk safety hurdle or it doesn't. Importantly, Arena, in its own regulatory filing, acknowledges that lorcaserin may indeed have a heart-valve safety problem. For more discussion on lorcaserin and the risk of valvulopathy, I suggest reading this old Arena column from Forbes' Matt Herper. Lastly, a brief discussion of lorcaserin's mediocre efficacy. Patients in the first two phase III studies (Bloom and Blossom) lost just 3.6% and 3.1% of their body weight (placebo adjusted) which falls short of one of two FDA-mandated efficacy guidelines for obesity drugs. Forty-seven percent of lorcaserin patients achieved a 5% weight loss in the two studies, which is double the weight loss achieved by placebo patients in one study (Bloom) but less than double in the second study (Blossom). On this basis alone, Arena had barely the efficacy data required to file lorcaserin for approval. FDA, in its first review, called lorcaserin's weight loss efficacy "marginal" and many of the advisory panel members concurred. The new Bloom-DM data don't help, particularly since this study enrolled patients who were obese and suffering from Type 2 diabetes. In other words, these patients more accurately represented the real world in which obese patients suffer from multiple weight-related health issues. The lorcaserin weight loss (placebo adjusted) in Bloom-DM was just 3%, the worst efficacy data from all three late-stage studies. Moreover, only 37% of lorcaserin patients achieved 5% weight loss -- again, the drug's worst performance in the three studies. Lorcaserin -- a weight loss drug that doesn't really help obese people lose weight, but which may exacerbate heart valve problems or fuel the growth of tumors. Not exactly the best risk-benefit equation. --Written by Adam Feuerstein in Boston. >To contact the writer of this article, click here: Adam Feuerstein. >To follow the writer on Twitter, go to http://twitter.com/adamfeuerstein.

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Arena Pharma's Own Data Point to Weight-Loss Drug Rejection

Some extreme brides-to-be are using feeding tubes to restrict their calorie intake and slim down before the big day.

Daniel Sheehan Photographers / Getty Images

How far would you go to lose weight?

Would you eat only through a feeding tube inserted in your nose? As unusual (and inconvenient) as that sounds, the feeding-tube diet, also known as the K-E diet, is the latest fad among brides who are looking to slim down before their big day.

The diet trend employs the same kind of feeding tube that doctors use in the hospital to nourish psychiatric patients who refuse to eat or those with physical conditions that prevent them from eating normally; coma and stroke patients depend on feeding tubes to survive, for instance.

(MORE:For Successful Weight Loss, Forget Fad Diets and Pills)

For weight-loss purposes, you dont need to be hospitalized, but you do have to live with a nasogastric tube inserted through your nose and threaded into your stomach. A protein pack feeds your body through the tube, by dispensing drops of a liquid mix of nutrients but no carbs totaling about 800 calories a day. Experts recommend that healthy adult women consume about 2,000 to 2,400 calories daily.

People use it drop significant amounts of weight or just to trim off a couple of extra pounds before a big event. At first I decided not to do it for people who just want to lose a few pounds, Dr. Oliver Di Pietro, who offers 10-day versions of the diet for $1,500 at his Bay Harbor Islands, Fla., office, told the New York Times. But then I thought, why should I say 5 or 10 pounds are not enough? People want to be perfect.

(MORE:Americans May Be Fatter than They Think, Study Says)

Medically speaking, the diet works by thrusting the body into the first stages of starvation. When you cut calories that drastically, the body responds by going into a state of ketosis, in which it starts to burn stored fat rather than consuming sugar to keep normal body functions going. In fact, each night, as you sleep, your body goes into a mild state of ketosis (which may explain in part why people who dont get enough sleep tend to be heavier). However, if this process is intentionally triggered, as with a severely calorie-restricted diet, the process can start to eat away at muscle.

See the original post here:
The Feeding-Tube Wedding Diet? Why It's a Bad Idea

Some extreme brides-to-be are using feeding tubes to restrict their calorie intake and slim down before the big day.

Daniel Sheehan Photographers / Getty Images

How far would you go to lose weight?

Would you eat only through a feeding tube inserted in your nose? As unusual (and inconvenient) as that sounds, the feeding-tube diet, also known as the K-E diet, is the latest fad among brides who are looking to slim down before their big day.

The diet trend employs the same kind of feeding tube that doctors use in the hospital to nourish psychiatric patients who refuse to eat or those with physical conditions that prevent them from eating normally; coma and stroke patients depend on feeding tubes to survive, for instance.

(MORE:For Successful Weight Loss, Forget Fad Diets and Pills)

For weight-loss purposes, you dont need to be hospitalized, but you do have to live with a nasogastric tube inserted through your nose and threaded into your stomach. A protein pack feeds your body through the tube, by dispensing drops of a liquid mix of nutrients but no carbs totaling about 800 calories a day. Experts recommend that healthy adult women consume about 2,000 to 2,400 calories daily.

People use it drop significant amounts of weight or just to trim off a couple of extra pounds before a big event. At first I decided not to do it for people who just want to lose a few pounds, Dr. Oliver Di Pietro, who offers 10-day versions of the diet for $1,500 at his Bay Harbor Islands, Fla., office, told the New York Times. But then I thought, why should I say 5 or 10 pounds are not enough? People want to be perfect.

(MORE:Americans May Be Fatter than They Think, Study Says)

Medically speaking, the diet works by thrusting the body into the first stages of starvation. When you cut calories that drastically, the body responds by going into a state of ketosis, in which it starts to burn stored fat rather than consuming sugar to keep normal body functions going. In fact, each night, as you sleep, your body goes into a mild state of ketosis (which may explain in part why people who dont get enough sleep tend to be heavier). However, if this process is intentionally triggered, as with a severely calorie-restricted diet, the process can start to eat away at muscle.

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With This Tube, I Thee Shed: What’s Wrong with the Latest Wedding Crash Diet?

Feeding-tube diet is latest weight-loss craze April 19th, 2012, 1:00 am posted by MARILYN KALFUS, THE ORANGE COUNTY REGISTER

Would you put a feeding tube in your nose for 10 days to lose weight? Thats being reported as the latest crash diet to hit the U.S., and its how some women are getting ready for their wedding photos.

Jessica Schnaider, courtesy of ABC News

BURN, BABY, BURN

Could you stick to an 800-a-day calorie for 10 days? How about by eating no food, and with a feeding tube hanging out of your nose?

The process, known as the K-E, or feeding-tube, diet, is being reported as an increasingly popular alternative to ordinary weight-loss programs.

From Good Morning America:

The program has dieters inserting a feeding tube into their nose that runs to the stomach. Theyre fed a constant slow drip of protein and fat, mixed with water, which contains zero carbohydrates and totals 800 calories a day. Body fat is burned off through a process called ketosis, which leaves muscle intact, Dr. Oliver Di Pietro of Bay Harbor Islands, Fla., said.

It is a hunger-free, effective way of dieting, Di Pietro said. Within a few hours and your hunger and appetite go away completely, so patients are actually not hungry at all for the whole 10 days. Thats what is so amazing about this diet.

Di Pietro says patients arent hospitalized. They wear the tube and carry the food solution around with them in a bag.

Excerpt from:
'Feeding-tube diet' is latest weight-loss craze

Spouses who snore may be spouses at risk.

But if you cant get your spouse to go to a sleep lab, theres another way.

Dr. Volkan Sumer of ICPR Family Practice, 1491 Valle Vista, Pekin, says his practice now offers the option of an in-home sleep evaluation as an alternative to spending the night in a sleep lab.

Sumer said, Snoring indicates a sleep disorder of some kind. If you are heavier, you have an increased chance of having sleep apnea. Obesity is becoming more of a problem in this country. One suggestion when people come in saying their husband or wife is really bothered by the spouses loud snoring, and the patient is overweight, is for them to lose weight. Snoring occurs when the pharynx falls back on itself and air passes through the small airway, and thats when you hear snoring. Air is being forced through to keep that tissue open, said Sumer.

People who suffer from sleep apnea also experience loud snoring that is interrupted by frequent episodes of obstructed breathing (or obstructive sleep apnea). Serious episodes can last more than 10 seconds and occur more than seven times an hour. That amounts to anywhere from 30 to 300 such events per night. Other symptoms of obstructive sleep apnea include always feeling tired, trouble concentrating or staying awake, waking up with headaches, waking up with a gasping sensation, waking up with a dry throat, experiencing restless sleep, depression, frequent trips to the bathroom at night, heartburn, excessive sweating at night, decreased libido, or rapid weight gain.

Sumer said that sleep apnea is directly correlated to diabetes. If sleep apnea goes undiagnosed it could lead to serious health risks including added risk levels for stroke and cardiac disorders. Episodes of obstructed breathing can also cause the heart to pump harder and reduces blood oxygen levels.

Sleepers go through cycles. Dr. Sarah Zallek is the medical director of the Illinois Neurological Institute Sleep Center at OSF Saint Francis, director of Illinois Neurological Institute Neurology at OSF Saint Francis, and a teaching physician at the University of Illinois College of Medicine.

Zallek said there are three stages of Non Rapid Eye Movement sleep. The first is considered light sleep. It is typically very short and occurs at the beginning of the night. Only 5 percent of sleep is spent in this stage. The second stage is part of light sleep but is deeper than the light sleep stage. A majority of NREM is spent in this stage.

The third stage is deep sleep. While someone is in this stage, it is hard to wake them. Less than 20 percent of time asleep is spent in deep sleep. Young children spend more time in deep sleep than adults do.

Rapid Eye Movement sleep is part of a healthy sleep cycle. REM is especially important for memory and cognition and without it we are not as able to process information. It is activated by new learning. It doesnt make us feel more awake, though. To be refreshed we need continuous sleep so we go through the sleep cycle. If you have sleep apnea, youll have interrupted sleep, said Zallek.

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How do you sleep at night? Alternative to sleep lab available in Pekin

When the burden of caring for others becomes too much to handle, the result can be more than just burnout.

Compassion fatigue can cause those in helping professions lose the ability to maintain a healthy balance of empathy and objectivity.

Psychologist Susan Fletcher will speak about compassion fatigue, also known as secondary traumatic stress disorder, at the Texas Tech University Health Sciences Center School of Nursing on April 2.

Fletcher, from Plano, said compassion fatigue happens to people in a helping profession when they dont take care of themselves. They tend to do more than burn out, she explained.

Burnout is obvious, and compassion fatigue is subtle.

Oftentimes, people experiencing compassion fatigue begin to make mistakes, she said.

Nurses could give the wrong dose of medicine, or physicians could read something wrong on a chart.

You know its not normal when it interferes with their personal life, she said. It interferes with their health, their personal life, their ability to do their job.

Compassion fatigue is common in professionals who are asked to do more with less, Fletcher explained. In the current state of the economy, she said, the susceptibility is startling.

Everyone in a helping profession is susceptible to compassion fatigue, from teachers, to administrators, to flight attendants, to financial advisors.

Read more:
Compassion fatigue may cripple health workers, expert says

When the burden of caring for others becomes too much to handle, the result can be more than just burnout.

Compassion fatigue can cause those in helping professions lose the ability to maintain a healthy balance of empathy and objectivity.

Psychologist Susan Fletcher will speak about compassion fatigue, also known as secondary traumatic stress disorder, at the Texas Tech University Health Sciences Center School of Nursing on April 2.

Fletcher, from Plano, said compassion fatigue happens to people in a helping profession when they dont take care of themselves. They tend to do more than burn out, she explained.

Burnout is obvious, and compassion fatigue is subtle.

Oftentimes, people experiencing compassion fatigue begin to make mistakes, she said.

Nurses could give the wrong dose of medicine, or physicians could read something wrong on a chart.

You know its not normal when it interferes with their personal life, she said. It interferes with their health, their personal life, their ability to do their job.

Compassion fatigue is common in professionals who are asked to do more with less, Fletcher explained. In the current state of the economy, she said, the susceptibility is startling.

Everyone in a helping profession is susceptible to compassion fatigue, from teachers, to administrators, to flight attendants, to financial advisors.

Read more:
Compassion fatigue may cripple health workers monhs agi

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